Amaninhis40swasadmittedafter4monthsofprogressivelegparesthesiasandsubjectivelegweakness.Theresultsofaneurologicalexaminationwereunremarkable.Theresultsofmagneticresonanceimaging(MRI)ofhisbrainandlaboratoryinvestigations,includingtestingforaquaporin-4antibodies,werenormal.Anexaminationofhiscerebrospinalfluidshowedmildhyperproteinorrachia(0.7g/L),pleocytosis(48leukocytes/mm3,95%ofwhichwereneutrophils),and3oligoclonalbands(mirrorpattern).MagneticresonanceimagingofhisspinedisclosedlongitudinallyextensivespinalcordlesionswithswellingatC2toC5andC7toD3levelsandnoncontiguous,patchygadoliniumenhancementalongthedorsalcolumns(Figure1).Theresultsofradiographyofhischestandhisserumangiotensinconvertingenzymelevelswerenormal.Computedtomographyofhischestrevealedhilarlymphadenopathywithincreaseduptakedetectedonfluorodeoxyglucose–positronemissiontomographicimages.Atransbronchialbiopsyrevealednoncaseatinggranulomasconsistentwithsarcoidosis(Figure2).Intravenousmethylprednisolonesodiumsuccinate(1g/dfor5days)wasadministered,withresolutionofsymptomsandnearly







































白癫风早期
白癫风早期


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